Search results for "Null allele"

showing 10 items of 15 documents

C4, BF, C3 Allele Distribution and Complement Activity in Healthy Aged People and Centenarians

1999

The aim of this study was to examine the complement system and the distribution of some human leukocyte antigen (HLA) class III alleles (C4, BF) in healthy aged people (77 centenarians and 89 elderly subjects). We have also studied the alleles of C3, a complement component genetically unrelated to HLA, the immunochemical levels of C4 and C3 and serum functional hemolytic activity for classical (CH50) and alternative (AP50) complement pathway. The levels of C3 and C4 and the CH50 and AP50 were found to be within the normal range. The frequencies of C3, BF, and C4A alleles were similar in the cohorts that have been studied. For C4B null allele (C4BQ0) a trend toward an increase in the older c…

AdultMaleAgingComplement Pathway AlternativeHuman leukocyte antigenBiologyComplement Hemolytic Activity AssayHemolysisComplement factor BCohort StudiesHLA AntigensHumansComplement Pathway ClassicalAlleleComplement ActivationAllelesAgedAged 80 and overPolymorphism GeneticC4AComplement C4Complement C3DNAMiddle AgedNull alleleComplement systemImmunologyCohortFemaleGeriatrics and GerontologyGene DeletionComplement Factor BThe Journals of Gerontology Series A: Biological Sciences and Medical Sciences
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Expression profiling of prospero in the Drosophila larval chemosensory organ: Between growth and outgrowth

2010

AbstractBackgroundThe antenno-maxilary complex (AMC) forms the chemosensory system of theDrosophilalarva and is involved in gustatory and olfactory perception. We have previously shown that a mutant allele of the homeodomain transcription factor Prospero (prosVoila1,V1), presents several developmental defects including abnormal growth and altered taste responses. In addition, many neural tracts connecting the AMC to the central nervous system (CNS) were affected. Our earlier reports on larval AMC did not argue in favour of a role ofprosin cell fate decision, but strongly suggested thatproscould be involved in the control of other aspect of neuronal development. In order to identify these fu…

Central Nervous SystemMESH : Transcription FactorsMESH: DrosophilaOF-FUNCTION SCREEN;MUSCA-DOMESTICA L;HOUSE-FLY LARVA;FINE-STRUCTURE;AXON GUIDANCE;TRANSCRIPTION FACTOR;PATTERN-FORMATION;GENETIC-ANALYSIS;NERVOUS-SYSTEMGenes InsectMESH: Genes InsectAXON GUIDANCEMUSCA-DOMESTICA L0302 clinical medicineMESH: Gene Expression Regulation DevelopmentalCluster AnalysisDrosophila ProteinsMESH: AnimalsTRANSCRIPTION FACTORMESH: Nerve Tissue ProteinsMESH : Nerve Tissue ProteinsOF-FUNCTION SCREENOligonucleotide Array Sequence AnalysisGenetics0303 health sciencesMESH : Central Nervous SystemMicrobiology and ParasitologyMESH : Genes InsectGene Expression Regulation DevelopmentalNuclear ProteinsMESH: Transcription FactorsNull alleleMicrobiologie et ParasitologieMESH : Oligonucleotide Array Sequence Analysis[ SDV.BBM.GTP ] Life Sciences [q-bio]/Biochemistry Molecular Biology/Genomics [q-bio.GN]Larva[SDV.BBM.GTP] Life Sciences [q-bio]/Biochemistry Molecular Biology/Genomics [q-bio.GN]DrosophilaDrosophila ProteinResearch ArticleBiotechnologylcsh:QH426-470MESH: Drosophila Proteinslcsh:BiotechnologyNerve Tissue ProteinsBiotechnologiesBiology03 medical and health sciencesMESH: Gene Expression ProfilingGENETIC-ANALYSIS[SDV.BBM.GTP]Life Sciences [q-bio]/Biochemistry Molecular Biology/Genomics [q-bio.GN]lcsh:TP248.13-248.65GeneticsAnimalsMESH : Cluster AnalysisMESH: Central Nervous SystemAlleleMESH : DrosophilaAlleles030304 developmental biologyMESH : LarvaMicroarray analysis techniquesHOUSE-FLY LARVAGene Expression ProfilingMESH : Gene Expression ProfilingMESH: AllelesWild typeMESH : Nuclear ProteinsProsperobiology.organism_classificationMESH : Drosophila ProteinsMESH: Cluster AnalysisNERVOUS-SYSTEMGene expression profilinglcsh:GeneticsMESH: Oligonucleotide Array Sequence AnalysisHomeoboxMESH : AnimalsMESH : Gene Expression Regulation DevelopmentalMESH : AllelesMESH: Nuclear ProteinsMESH: Larva030217 neurology & neurosurgeryTranscription FactorsPATTERN-FORMATIONFINE-STRUCTURE
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Genetic basis of human complement C4A deficiency. Detection of a point mutation leading to nonexpression.

1993

Abstract The fourth component of the human complement system (C4) is coded for by two genes, C4A and C4B, located within the MHC. Null alleles of C4 (C4Q0) are defined by the absence of C4 protein in plasma. These null alleles are due either to large gene deletions or to nonexpression of the respective genes. In a previous study, evidence was obtained for nonexpressed defective genes at the C4A locus, and for gene conversion at the C4B locus. To further characterize the molecular basis of these non-expressed C4A genes, we selected nine pairs of PCR primers from flanking genomic intron sequences to amplify all 41 exons from individuals with a defective C4A gene. The amplified products were s…

ElectrophoresisMolecular Sequence DataLocus (genetics)BiologyPolymerase Chain ReactionAutoimmune DiseasesHumansPoint MutationGene conversionAmino Acid SequenceGeneGeneticsPolymorphism GeneticBase SequenceHaplotypeC4AGene AmplificationImmunologic Deficiency SyndromesComplement C4aSingle-strand conformation polymorphismGeneral MedicineExonsSequence Analysis DNAMolecular biologyNull alleleStop codonHaplotypesResearch Article
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Genetic characterisation of CSN2 gene in Girgentana goat breed

2014

Among calcium sensitive caseins, β-casein is the most abundant in goat milk, representing up to 50% of total casein content. The goat β-casein locus has been widely investigated and at least ten alleles have been identified in different goat breeds. The aim of this work was to investigate the polymorphisms of β-casein gene in Girgentana dairy goat breed in order to assess the genotype distribution and evaluate how frequencies have changed during the last 10 years, as genotype is known to influence technological and nutritional milk properties. Sequencing analysis and alignment of the obtained sequences of β-casein exon 7, showed the presence of C, C1, and A strong alleles, and 0' null allel…

Genetics040301 veterinary sciences0402 animal and dairy scienceCSN2 locus Genetic polymorphisms Genetic evolution Girgentana goat breedLocus (genetics)04 agricultural and veterinary sciencesBiology040201 dairy & animal scienceNull alleleBreed0403 veterinary scienceExonSettore AGR/17 - Zootecnica Generale E Miglioramento GeneticoCaseinGenotypeAnimal Science and Zoologylcsh:Animal cultureAlleleGenelcsh:SF1-1100
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Primers for nine microsatellite loci in the hermaphroditic snailLymnaea stagnalis

2003

Variation in and amplification conditions for nine polymorphic microsatellite loci identified from Lymnaea stagnalis , a hermaphroditic pulmonate snail, are described. Eight populations from central Finland were studied, which varied in terms of both observed polymorphism and heterozygosity. The number of alleles at each locus is moderate (two to seven), except for one exceptional locus having 16 alleles, and for which null alleles are possible. There is no evidence for genotypic disequilibrium in the populations for all pairs of loci. Heterozygosity levels are indicative of outcrossing in L. stagnalis , whose mating system will be characterized further using these markers.

GeneticsEcologybiologySelfingLocus (genetics)Lymnaea stagnalisbiology.organism_classificationBiochemistryNull alleleGeneral Biochemistry Genetics and Molecular BiologyLoss of heterozygosityGenotypeMicrosatelliteAlleleMolecular Ecology Notes
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New alleles and mutational events at 14 STR loci from different German populations.

2007

The molecular origin of DNA mutations and the mutation rates were analyzed at 14 short tandem repeat (STR) loci with samples from trio cases derived from 10 different German population samples. STR loci comprised of D2S1360, D3S1744, D4S2366, D5S2500, D6S474, D7S1517, D8S1132, D10S2325, D12S391, D18S51, D19S246, D20S480, D21S226, and D22S689. In a total of 488 meioses, 16 isolated genetic inconsistencies in 8 different STRs were observed, whereas no mutations were found at the other loci. The data of five mutations suggested the presence of silent or null alleles due to sequence variation in primer binding site. This could be confirmed for four suspected cases by the use of alternative prim…

GeneticsForensic GeneticsMaleMutation rateBase SequenceSTR multiplex systemDNABiologyNull allelePathology and Forensic MedicineGenetics PopulationSTR analysisGene FrequencyGermanyMutationGeneticsMicrosatelliteHumansFemalePrimer (molecular biology)AllelePrimer binding siteAllelesDNA PrimersMicrosatellite RepeatsForensic science international. Genetics
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Isolation and characterization of 10 highly polymorphic di- and trinucleotide microsatellite markers in the mayfly Ameletus inopinatus (Ephemeroptera…

2008

We describe the isolation of ten polymorphic microsatellite loci from the mayfly Ameletus inopinatus. Loci had di- or trinucleotide repeat motifs and were highly variable with three to 17 alleles (mean = 7.15). Observed heterozygosity ranged from 0.143 to 0.905. One locus (Ami_202) showed significant deviation from Hardy–Weinberg equilibrium in one population, but no evidence for null alleles. One locus (Ami_73) was significantly linked with three other loci. The remaining nine loci should prove highly informative for population genetic studies.

Geneticseducation.field_of_studyPopulationLocus (genetics)Biologybiology.organism_classificationNull alleleLoss of heterozygosityMayflyGeneticsMicrosatelliteeducationTrinucleotide repeat expansionMicrosatellite enrichmentEcology Evolution Behavior and SystematicsBiotechnologyMolecular ecology resources
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Isolation and characterization of 11 polymorphic trinucleotide microsatellite markers in the stonefly Arcynopteryx compacta (Plecoptera: Perlodidae).

2011

We describe the isolation of 11 polymorphic trinucleotide microsatellite loci from the stonefly Arcynopteryx compacta. Loci were highly variable with 3 to 14 alleles (mean = 6.45). Observed heterozygosity ranged from 0 to 0.867. Seven loci showed significant deviation from Hardy–Weinberg equilibrium across both populations. There was no evidence for null alleles, and thus, Hardy–Weinberg departures could have resulted from genetic structure between populations or subpopulations. No linkage between loci was found. The 11 loci should prove highly informative for population genetic studies.

Geneticseducation.field_of_studybiologyPopulationbiology.organism_classificationNull alleleLoss of heterozygosityPerlodidaeGenetic structureGeneticsMicrosatelliteAlleleeducationMicrosatellite enrichmentEcology Evolution Behavior and SystematicsBiotechnologyMolecular ecology resources
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Disruption of TCBA1 associated with a de novo t(1;6)(q32.2;q22.3) presenting in a child with developmental delay and recurrent infections

2005

A boy with developmental delay, particularly of speech, a distinct face, antineutrophil cytoplasmic antibodies, and recurrent infections was found to have an apparently balanced de novo t(1;6)(q32.3;q22.3) translocation. Fluorescent in situ hybridisation with BAC/PAC clones and long range polymerase chain reaction products assessed in the human genome sequence localised the chromosome 1 breakpoint to a 9.8 kb segment within a hypothetical gene, LOC388735, and the chromosome 6 breakpoint to a 12.8 kb segment in intron 4 of the T-cell lymphoma breakpoint-associated target 1 (TCBA1) gene. Disruption and/or formation of TCBA1 fusion genes in T cell lymphoma and leukaemia cell lines suggests a r…

Malemedicine.medical_specialtyDevelopmental DisabilitiesMolecular Sequence DataShort ReportBiologyInfectionsTranslocation GeneticFusion geneExonMiceMolecular geneticsGeneticsmedicineAnimalsHumansAmino Acid SequenceChildGeneGenetics (clinical)GeneticsGenome HumanGene Expression ProfilingBreakpointChromosome MappingMembrane ProteinsChromosome BreakageExonsNull alleleMolecular biologyGene expression profilingChromosomes Human Pair 1Child PreschoolCytogenetic AnalysisChromosomes Human Pair 6Chromosome breakage
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Null alleles of human complement C4. Evidence for pseudogenes at the C4A locus and for gene conversion at the C4B locus

1990

The two genes for the C4A and C4B isotypes of the fourth component of human complement are located in the MHC class III region. Previous studies have demonstrated the unusual expression of C4 genes in the form of aberrant or duplicated haplotypes. Null alleles of C4A or C4B (AQ0 or BQ0) have been defined by the absence of gene products and occur at frequencies of 0.1-0.3. However, only some C4 null alleles are due to gene deletions, the remainder were thought to be nonexpressed genes. We have analyzed the C4 gene structure of 26 individuals lacking either C4A or C4B protein. The DNA of individuals with apparently nonexpressed C4 genes was tested for the presence of C4A- and C4B-specific seq…

PseudogeneImmunologyMolecular Sequence DataGene ConversionLocus (genetics)chemical and pharmacologic phenomenaPolymerase Chain ReactionRestriction fragmentComplement C4bImmunology and AllergyHumansGene conversionAlleleGeneAllelesGeneticsbiologyBase SequenceHomozygoteC4AComplement C4aComplement C4ArticlesDNANull alleleMolecular biologyGenesbiology.proteinDNA ProbesOligonucleotide ProbesPseudogenesThe Journal of Experimental Medicine
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